Volume 2 | Issue 1 | DOI: https://doi.org/10.33696/cardiology.2.006
Hypertrophic Cardiomyopathy and the Troponins: The Enigma Remains
- 1Janson Precision and Regenerative Medicine at MIT, The Koch Institute for Integrative cancer research, Massachusetts Institute of Technology, 500, Main street, Cambridge, MA, USA
Rameen Shakur, email@example.com
Received Date: January 11, 2021
Accepted Date: March 10, 2021
Shakur R. Hypertrophic Cardiomyopathy and the Troponins: The Enigma Remains. J Clin Cardiol. 2021; 2(1):1-5.
Copyright: © 2021 Shakur R. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
The differential diagnosis of a mediastinal mass includes benign and malignant etiologies like lymphomas, thymic tumors, thyroid goiter, infections such as tuberculosis, chronic granulomatous disorders such as sarcoidosis, germ cell tumors, among others. Based on its location, they can be classified as anterior, medium or posterior.
Hypertrophic cardiomyopathy (HCM) is a heart muscle disorder and is the most common form of Mendelianinherited heart disease, affecting approximately 0.2% of the global population. In adults the disease is often inherited as an autosomal dominant trait caused by mutations, mainly in one of the 23 cardiac sarcomere protein genes.
A 59-year-old man presents to the hospital following a syncopal episode after a low-speed car accident without visible injuries. He was diagnosed with stress cardiomyopathy and severe triple-vessel coronary artery disease. The case was complicated by complete heart block that culminated in refractory cardiogenic shock. Literature and current approach are reviewed.
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